Consumption, phthisis, the white plague—a killer by any other name, tuberculosis (TB) has stalked humanity since at least the ...

Tuberculosis bacteria rely on a family of genes that help them survive the challenging journey from one person's lungs to another person's during coughing, sneezing or talking, according to ...

When German biologist and doctor Robert Koch announced in 1882 that he had discovered the bacterial cause of tuberculosis, he said, "If the number of victims which a disease claims is the measure ...

However, as a bacterial colony develops, “growing pains” are inevitable, as proliferating cells must balance growth rates with the stress of overcrowding and nutrient resource limitations. Other ...

with a relative of TB bacteria detected since the dinosaur age, and an ancestor having infected our early human ancestors. Indeed, the pathogen seems like ancient history — but as recent ...

They engineered a strain of M. tuberculosis carrying a triple kill switch, which uses three independent molecular mechanisms to kill the bacteria. Even in severely immunocompromised mice ...

They were able to show that BTZ-043 effectively penetrates TB lesions and accumulates there in high concentrations. Consequently, the drug can fight Mtb bacteria even in hard-to-reach areas.

J Midwifery Womens Health. 2007;52(4):415-416. Tuberculosis (TB) is an infection caused by bacteria. It usually affects the lungs, but it can also cause problems in other parts of the body.

They engineered a strain of M. tuberculosis carrying a triple kill switch, which uses three independent molecular mechanisms to kill the bacteria. Even in severely immunocompromised mice ...

Still, for decades, the fight against TB has been frozen in time, even as the bacteria that causes it has become more resistant. The world’s only TB vaccine protects young infants and children but ...

Engineered Mycobacterium tuberculosis triple-kill-switch strain provides controlled tuberculosis infection in animal models. Nature Microbiology , 2025; 10 (2): 482 DOI: 10.1038/s41564-024-01913-5 ...